Acyclovir resistant hsv treatment
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the treatment of acyclovir-resistant mucocutaneous HSV infections in immunocompromised patients. According to IQVIA, US sales of Foscarnet
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For HSV that is resistant to acyclovir or ganciclovir, cidofovir and foscarnet are potential alternative treatments. These drugs do not require phosphorylation
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mechanism of action as acyclovir. Thus, HSV isolates resistant to acyclovir are also resistant to valacyclovir, famciclovir, and penciclovir. (See
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Acyclovir resistance amongst immunocompetent patients with HSV keratitis can be as high as 6.4%. 200, 201 Resistance to acyclovir may occur more commonly in patients with recurrent ocular HSV infections. 200 Compared to patients with HSV keratitis caused by an HSV isolate sensitive to acyclovir, patients infected by an acyclovir-resistant
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Patients with a TK mutation causing acyclovir resistance can be treated with systemic foscarnet. 200, 201 An HSV resistant to both foscarnet and acyclovir can be effectively treated with cidofovir. The role of topical trifluridine in the treatment of acyclovir-resistant, mucocutaneous HSV seemed promising in a pilot study conducted in
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Acyclovir-resistant HSV infections. Acyclovir-resistant HSV infections are often seen in immunocompromised patients (eg, patients with HIV infection). Resistant isolates result in severe, debilitating mucosal disease, and visceral dissemination.
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General Information ; Lab Name: HSV Acyclovir Drug Resistance ; Lab Code: HSVDR ; Epic Ordering: HSV Acyclovir Drug Resistance ; Description. HSV Type 1 and HSV
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by L Szenborn 2024 Cited by 21As a result, effective acyclovir treatment is under- prescribed in In practice, transmission of acyclovir-resistant HSV and. VZV
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mechanism of action as acyclovir. Thus, HSV isolates resistant to acyclovir are also resistant to valacyclovir, famciclovir, and penciclovir.
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Gavin explains , For instance, an antibiotic, if you take the same antibiotic every infection, your body will stop responding to it.
Antibiotics stop working because the dose is too low or the treamement is too short a time not because of frequent use. During treatment, the most resistant bacteria are the last to die so any bacteria that survive a course of treatment are now selected to build the next generation of stronger and more resistant bacteria.
The solution is exactly the opposite of Gavin's statement i.e. more antibiotic for a longer time to ensure all bacteria are killed.